Lumbar Trauma: Trauma to the lumbar area of the back can be another cause of neuropathy and chronic pain. This trauma can be as slight as lifting a bag of groceries out of the trunk, picking up a grandchild, or bending down to tie a shoe. Our studies show a 60% correlation between repeated injuries to the lower back and subsequent development of neuropathy and chronic pain symptoms.
During the acute phase of localized trauma, inflammation develops reducing arterial and venous blood to the lumbar synaptic junctions. Nerves in the region temporarily shrink due to the reduction in activity. Since the body tends to conserve resources, the affected nerves begin to atrophy, the synaptic junction gap begins to widen, and synaptic minerals leech away making signal transmission more difficult.
Signals of normal strength can no longer cross synapses that are damaged by the reduction in blood flow. The loss of signals across the synapses compounds the process of deterioration. Muscle atrophy and a host of other problems follow. We have found that a signal delivered at 7.83 cycles per second (the body’s natural electromagnetic resonant frequency) and at an amplitude approximately 10 times that originally required will cross these enlarged synapses, repolarize them.
High Blood Pressure Medication: High blood pressure medication not only lowers blood pressure, it also reduces the ability of the arterial blood to refill the veins. This vacancy results as the venous muscle pumps the blood back to the heart. When this occurs the blood has a tendency to pool in the lower extremities; the nerves and synaptic junctions do not have enough necessary nutrition and oxygen to maintain their health resulting in nerve cell atrophy, loss of mineralization, and conductivity of the synaptic junctions as explained above.